By M.M. Hirschler
This e-book summarizes a entire examine of the significance of carbon monoxide [CO] in hearth atmospheres. it will likely be correct to associations enthusiastic about fireplace examine, to industries generating flamable fabrics and to these excited by hearth from security and clinical standpoints.
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Additional info for Carbon Monoxide and Human Lethality: Fire and Non-Fire Studies
34 The contribution of inhaled soots and adsorbed materials or of irritating gases, such as a aldehydes or hydrochloric acid, was not known. Heavy concentrations of inorganic metals (lead, antimony, and others) and adsorbed pulmonary irritants were observed in the soots that were deposited throughout the trachea and lung tissues. Acetaldehyde was also recovered from lung specimens Carbon monoxide and human lethality 38 FIGURE 14. Medical causes leading to fire fatalities. 76 of fire victims. The contributions of the soots and adsorbed materials to breathing difficulties (such as lung edema) are likely to be considerable, but their specific contributions to fatalities were not clear.
In both cases, at the end of exposure the same level of blood carboxyhemoglobin (about 50%) was reached. The biochemical determinations in the blood (pH, glucose, lactate, pyruvate) and brain tissue (lactate, pyruvate) were carried out immediately after termination of the exposure and after periods of recovery. CO exposure resulted in a decreased blood pH, increased level of blood glucose, as well as that of lactate and pyruvate both in blood and brain tissue. These changes were much more pronounced following the “longer-lesser” exposure than after the “shorter-intense” one, although blood concentrations of COHb were the same.
The uppermost curve (IV) represents the approximate threshold at which rapidly fatal systemic hyperthermia occurred. 87 The most constant postmortem finding in animals that died of hyperthermic shock within thirty minutes of the exposure was the presence of hemorrhages throughout the internal viscera. 87 It is interesting to note that pigs showed little pulmonary edema even on exposure to very hot gases. 5°C or higher. No pig whose rectal temperature rose above 44°C survived more than a few minutes.