Cardiovascular Soft Tissue Mechanics by Gerhard A. Holzapfel, Thomas C. Gasser (auth.), Stephen C.

By Gerhard A. Holzapfel, Thomas C. Gasser (auth.), Stephen C. Cowin, Jay D. Humphrey (eds.)

This unique quantity of the magazine of Elasticity represents the 1st in a brand new p- gram devoted to the occasional book of collections of invited, reviewed papers of topical curiosity. the aim of this software is to highlight the dev- opments and purposes within the mechanics of fabrics inside particular components that could improve progress and supply perception for the development of the sector in addition to advertise basic figuring out and easy discovery. gentle Tissue Mechanics is a space of biomechanics that attracts seriously upon f- damental principles and fabric types from nonlinear elasticity and viscoelasticity. a huge aim of this learn is to appreciate these mechanics houses of middle, artery, collagen and skeletal muscle tissues that may be used for the prognosis of illnesses and the advance of human existence. This quantity illustrates how test, modeling and computation is at present hired during this rising box. may well 2001 ROGER FOSDICK Editor-in-Chief magazine of Elasticity sixty one: ix–xii, 2000. ix Preface There are basic parts for the applying of elasticity within the biomechanics of tissues: not easy tissue mechanics (e.g., bone, the teeth, horns, etc.) and delicate tissue - chanics (e.g., epidermis, tendons, arteries, etc.). The distinguishing function among those tissue forms is the quantity of physiological “normal” deformation they event. whereas “hard” tissues basically adventure small deformations, smooth tissues usually event huge deformations. From a biomechanics standpoint gentle tissues fall in the realm of finite elasticity.

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19. 20. 21. 22. 23. 24. 25. 26. 27. 28. 29. ARTERIAL WALL MECHANICS 30. 31. 32. 33. 34. 35. 36. 37. 38. 39. 40. 41. 42. 43. 44. 45. 46. 47. 48. 49. 50. 51. 52. 53. H. Hoppmann and L. Wan, Large deformation of elastic tubes. J. Biomech. 3 (1970) 593– 600. R. Hughes, The Finite Element Method: Linear Static and Dynamic Finite Element Analysis, Prentice-Hall, Englewood Cliffs, NJ (1987). D. Humphrey, Mechanics of arterial wall: Review and directions. Critical Reviews in Biomed. Engr. 23 (1995) 1–162.

Types III and V) within the cerebral vasculature, which in turn may weaken the arterial wall [41, 74]. Similarly, it has been hypothesized that an asymmetrically formed circle of Willis may be of genetic origin, and may increase the hemodynamic load on portions of the vasculature [20, 92]. There is a pressing need for much more research on the roles of genetics, risk factors, cellular responses to mechanical stresses, and hemodynamics in the pathogenesis. Aneurysms typically enlarge from the initially small out-pouching or dilatation of the arterial wall, which can result in lesions having diameters up to 30 mm as well as complex shapes and composition.

Engr. 113 (1991) 42–55. C. C. Fung, Species dependence of the zero–stress state of aorta: Pig versus rat. J. Biomech. Engr. 113 (1991) 446–451. K. Hayashi, Experimental approaches on measuring the mechanical properties and constitutive laws of arterial walls. J. Biomech. Engr. 115 (1993) 481–488. A. Holzapfel, Nonlinear Solid Mechanics. A Continuum Approach for Engineering, Wiley, Chichester (2000). A. C. Gasser, A viscoelastic model for fiber-reinforced composites at finite strains: Continuum basis, computational aspects and applications.

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