Cholestasis - A Medical Dictionary, Bibliography, and by Icon Health Publications

By Icon Health Publications

This can be a 3-in-1 reference booklet. It supplies an entire scientific dictionary protecting thousands of phrases and expressions in relation to cholestasis. It additionally offers huge lists of bibliographic citations. ultimately, it offers info to clients on how you can replace their wisdom utilizing a variety of web assets. The publication is designed for physicians, scientific scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to get to grips with study devoted to cholestasis. in the event that your time is effective, this ebook is for you. First, you won't waste time looking the net whereas lacking loads of suitable info. moment, the publication additionally saves you time indexing and defining entries. eventually, you won't waste time and cash printing 1000's of web content.

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Children's Hospital of Philadelphia 34Th St and Civic Ctr Blvd Philadelphia, Pa 191044399 Timing: Fiscal Year 2002; Project Start 15-AUG-2000; Project End 30-JUN-2005 Summary: Disorders of bile duct development constitute a major cause of morbidity and mortality in the pediatric population, yet little is understood regarding the genetic signaling and molecular control of bile duct formation during development. The gene Jagged1, encoding a ligand in the Notch intercellular signaling pathway, has recently been found to be involved in this process.

D. are evaluating the role of secretory lineages during intestinal adapation, and have demonstrated an early increased production of intestinal secretory lineages occurs following resection. I hypothesize that the early increased production of intestinal secretory lineages following massive intestinal loss is essential to amplify the early signals initiating intestinal adaptation. This grant proposes the use of a specific progenitor assay somatic mutation (SPASM) sytem to identify dynamic changes within intestinal progenitor compartments that occur following a 50% intestinal resection in mice.

The relationship between Mrp4 regulation by the second messenger cAMP and the relation with proliferation will be further explored in Aim 1 of the current application. Because our results show that Mrp4 has a role in the transport of cholestatic conjugated bile salts and is upregulated under cholestatic conditions, I will determine if Mrp4 absence renders the liver more susceptible to cholestatic injury. I will also use the Mrp4 -/- animals to identify mechanisms (Aim II) in the liver that compensate under cholestasis, but are independent of Mrp4.

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