Diabetic Retinopathy - A Medical Dictionary, Bibliography, by Icon Health Publications

By Icon Health Publications

In March 2001, the nationwide Institutes of wellbeing and fitness issued the next caution: "The variety of websites providing health-related assets grows each day. Many websites supply helpful details, whereas others could have details that's unreliable or misleading." in addition, as a result swift raise in Internet-based info, many hours could be wasted looking, picking out, and printing.This e-book was once created for doctors, scholars, and individuals of most people who are looking to behavior clinical learn utilizing the main complicated instruments to be had and spending the smallest amount of time doing so.

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Extra info for Diabetic Retinopathy - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References

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In these patients there is pathologic growth of new blood vessels in the inner retina. These poorly formed blood vessels burst, bleeding into the retina and obscuring vision. Current treatment for these patients is to detect the neovascular formations and ablate tissue around the vessels, intending to reduce or eliminate further angiogenic response. It is proposed that new blood vessels are formed Studies 31 in response to local tissue hypoxia, but it is not possible at this time to identify focal areas of hypoxia in the retina.

Good evidence suggests that photoreceptor degeneration in this model occurs through apoptosis. The ability of anti-apoptotic genes expressed in transgenic mice to inhibit photic injury induced cell death will be tested. Specifically, the ability of anti-oxidant genes and antiapoptosis genes that act upstream or downstream in apoptosis pathways to inhibit photoreceptor degeneration will be evaluated. Further, the intracellular localization of cytochrome c, a mediator of apoptosis, and the role of caspase activation will be probed.

Capillary ischemia could be due at least in part to obstruction caused by abnormal or accelerated growth of vascular smooth muscle cells. The increases in angiotensin (Ang) II and insulin levels that occur in hypertension and noninsulin dependent diabetes (type 2) may contribute to this process within the small ophthalmic arteries. Thus, insulin and Ang II could act in a synergistic manner. Inhibiting the renin-angiotensin system (RAS) confers a therapeutic benefit in the treatment of diabetic retinopathy.

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